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New Microsoft Syntex Freeform Document Processing Model & Syntex Model  Renames - Leon Armston's Blog
New Microsoft Syntex Freeform Document Processing Model & Syntex Model Renames - Leon Armston's Blog

Insects | Free Full-Text | CRISPR/Cas9-Mediated Mutagenesis of Abdominal-A  and Ultrabithorax in the Asian Corn Borer, Ostrinia furnacalis
Insects | Free Full-Text | CRISPR/Cas9-Mediated Mutagenesis of Abdominal-A and Ultrabithorax in the Asian Corn Borer, Ostrinia furnacalis

Application of CRISPR-Cas9-Mediated Genome Editing for the Treatment of  Myotonic Dystrophy Type 1: Molecular Therapy
Application of CRISPR-Cas9-Mediated Genome Editing for the Treatment of Myotonic Dystrophy Type 1: Molecular Therapy

Small Molecule Screening Discovers Compounds that Reduce FMRpolyG Protein  Aggregates and Splicing Defect Toxicity in Fragile X-Associated  Tremor/Ataxia Syndrome | SpringerLink
Small Molecule Screening Discovers Compounds that Reduce FMRpolyG Protein Aggregates and Splicing Defect Toxicity in Fragile X-Associated Tremor/Ataxia Syndrome | SpringerLink

HAP40 is a conserved central regulator of Huntingtin and a potential  modulator of Huntington's disease pathogenesis | PLOS Genetics
HAP40 is a conserved central regulator of Huntingtin and a potential modulator of Huntington's disease pathogenesis | PLOS Genetics

The good, the bad, and the ugly: Evolutionary and pathological aspects of  gene dosage alterations | PLOS Genetics
The good, the bad, and the ugly: Evolutionary and pathological aspects of gene dosage alterations | PLOS Genetics

An Intein-based Strategy for the Production of Tag-free Huntingtin Exon 1  Proteins Enables New Insights into the Polyglutamine Dependence of Httex1  Aggregation and Fibril Formation - ScienceDirect
An Intein-based Strategy for the Production of Tag-free Huntingtin Exon 1 Proteins Enables New Insights into the Polyglutamine Dependence of Httex1 Aggregation and Fibril Formation - ScienceDirect

Dynamics of the Proline-Rich C-Terminus of Huntingtin Exon-1 Fibrils. -  Abstract - Europe PMC
Dynamics of the Proline-Rich C-Terminus of Huntingtin Exon-1 Fibrils. - Abstract - Europe PMC

Dynamics of the Proline-Rich C-Terminus of Huntingtin Exon-1 Fibrils | The  Journal of Physical Chemistry B
Dynamics of the Proline-Rich C-Terminus of Huntingtin Exon-1 Fibrils | The Journal of Physical Chemistry B

Site‐Specific Phosphorylation of Huntingtin Exon 1 Recombinant Proteins  Enabled by the Discovery of Novel Kinases - Chiki - 2021 - ChemBioChem -  Wiley Online Library
Site‐Specific Phosphorylation of Huntingtin Exon 1 Recombinant Proteins Enabled by the Discovery of Novel Kinases - Chiki - 2021 - ChemBioChem - Wiley Online Library

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A cell cycle-independent, conditional gene inactivation strategy for  differentially tagging wild-type and mutant cells | eLife
A cell cycle-independent, conditional gene inactivation strategy for differentially tagging wild-type and mutant cells | eLife

In heart failure reactivation of RNA-binding proteins is associated with  the expression of 1,523 fetal-specific isoforms | PLOS Computational Biology
In heart failure reactivation of RNA-binding proteins is associated with the expression of 1,523 fetal-specific isoforms | PLOS Computational Biology

Heterogeneous Nuclear Ribonucleoproteins: Implications in Neurological  Diseases | SpringerLink
Heterogeneous Nuclear Ribonucleoproteins: Implications in Neurological Diseases | SpringerLink

Genes containing hexanucleotide repeats resembling C9ORF72 and expressed in  the central nervous system are frequent in the human genome - ScienceDirect
Genes containing hexanucleotide repeats resembling C9ORF72 and expressed in the central nervous system are frequent in the human genome - ScienceDirect

IJMS | Free Full-Text | A Novel Huntington’s Disease Assessment  Platform to Support Future Drug Discovery and Development
IJMS | Free Full-Text | A Novel Huntington’s Disease Assessment Platform to Support Future Drug Discovery and Development

RNA toxicity in non‐coding repeat expansion disorders | The EMBO Journal
RNA toxicity in non‐coding repeat expansion disorders | The EMBO Journal

Sam68 sequestration and partial loss of function are associated with  splicing alterations in FXTAS patients | The EMBO Journal
Sam68 sequestration and partial loss of function are associated with splicing alterations in FXTAS patients | The EMBO Journal